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Guenther, Paul This work focuses on modeling of the effects of ionizing radiation on cells, primarily on, the influence of the DNA repair pathway availability and the radiation quality on the cell-survival probability.

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The radiation quality manifests itself in the microscopic ionization pattern. Both models assume that cell survival can be modeled based on the spatial distribution of Double-Strand Breaks DSB of the DNA damage patternwithin a higher order chromatin structure.

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One central assumption of the LEM is that the same damage patterns imply same effects, regardless of the radiation quality. In order to predict the damage pattern the microscopic local dose distribution of ions, described by the amorphous track structure, is evaluated. The cell survival after ion irradiation is predicted from a comparison with corresponding damage patterns after photon irradiation.

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The cell-survival curves after high dose photon irradiation cannot be predicted from the Linear Quadratic LQ Model due to their transition towards a linear dose dependence. This work uses the GLOBLE-Model to introduce a novel mechanistic approach, which allows the threshold dose to be predicted for optionen kompletter kurs für profis simon vine transition from a linear quadratic dose dependence, of survival curves at low doses, to a linear dose dependence at high doses.

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Furthermore, a method is presented, which allows LEM to predict the survival of synchronous cells after ion irradiation based on the cell survival after photon irradiation. This method is able to qualitatively predict the influence of the cell-cycle and the radiation quality on radiosensitivity.

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The first approach predicts the RBE based on the survival of asynchronous cells. The second approach predicts the RBE from the sum of survival curves of the subpopulations, which contribute to the asynchronous cell population. Both approaches lead to qualitatively similar results.

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In the context of describing the microscopic dose deposition of ion irradiation by the amorphous track structure, two questions are addressed: 1. Is it possible to improve the prediction of cell survival after ion irradiation by a more sophisticated composition of the direct and indirect effect?

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Regarding the first question, it is shown that a more detailed evaluation of the direct and indirect effect improves the agreement of the LEM predictions to data. Concerning the second question, it is shown that the amorphous track structure can be used for the prediction of DNA damage induction on the nm-scale similar to an ab initio Monte Carlo simulation. In the context of cell-survival modeling, the relevant length scale for DNA damage-clustering is often discussed.

Deutsches Institut für Normung. All patients but one recovered from symptoms after core decompression. Although the signal morphology of the necrotic area remained unchanged in the majority of the cases, a decrease of the joint effusion was observed as well as an ongoing signal increase after gadopentetate dimeglumine application.

There are also models which predict the cell survival after ion irradiation based on complex DNA-damage-clusters on the nm-scale. Therefore, it is possible to predict the cell survival after ion irradiation based on both scales. However, the cell survival after photon irradiation cannot be predicted based on damage-clusters on the nm-scale.

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